Description
Springer Physiology and Biochemistry of Plant-Pathogen Interactions 1st Editon 2012 Softbound by I. J. Misaghi
There has been a significant surge of interest in the study of the physiology and biochemistry of plant host-parasite interactions in recent years, as evidenced by the number of research papers currently being published on the subject. The in creased interest is probably based on the evidence that effective management of many plant diseases is, for the most part, contingent upon a clear understanding of the nature of host-parasite interactions. This intensified research effort calls for a greater number of books, such as this one, designed to compile, synthesize, and evaluate widely scattered pieces of information on this subject. The study of host-parasite interactions concerns the struggle between plants and pathogens, which has been incessant throughout their coevolution. Such in teractions are often highly complex. Pathogens have developed sophisticated of fensive systems to parasitize plants, while plants have evolved diversified defen sive strategies to ward off potential pathogens. In certain cases, the outcome of a specific host-parasite interaction seems to depend upon the presence or efficacy of the plant's defense system. A plant may become diseased when a parasite manages to invade it, unhindered by preexisting defense systems and/or without eliciting the plant's induced resistance response(s). Absence of disease may re flect the inability of the invading pathogen to overcome the plant's defense sys tem(s). 1 Definition.- I. Introduction.- II. Definition of a Few Terms.- 2 Attraction to and Penetration of Plants by Pathogens.- I. Introduction.- II. Attraction of Pathogens to Plants.- III. Penetration.- IV. Factors Influencing Attraction, Penetration, and Infection Processes.- 3 The Role of Pathogen-Produced Cell-Wall-Degrading Enzymes in Pathogenesis.- I. Introduction.- II. Structure and Chemistry of the Cell Wall.- III. Pathogen-Produced Cell-Wall-Degrading Enzymes.- 4 The Role of Pathogen-Produced Toxins in Pathogenesis.- I. Introduction.- II. Definition and Classification of Pathogen-Produced Toxins.- III. Toxin Categories.- IV. Evaluation of the Criteria Used to Establish the Role of Toxins in Pathogenesis.- V. Host-Selective Toxins.- VI. Nonselective Toxins.- VII. Mode of Action of Toxins.- VIII. Toxin Binding Sites.- IX. Mechanism of Plant Resistance to Toxin-Producing Pathogens.- 5 Alterations in Permeability Caused by Disease.- I. Introduction.- II. Composition and Structure of Membranes.- III. Cell Permeability Theories.- IV. Changes in Cell Permeability Caused by Disease.- V. Mechanisms of Permeability Changes in Diseased Plants.- 6 Alterations in Water Relations Caused by Disease.- I. Introduction.- II. Definition of a Few Terms.- III. Water Movement in Plants.- IV. Transpiration.- V. Disruption of Membrane Permeability in Leaf Cells by Pathogen-Produced Substances.- VI. Water Potential and Disease Severity.- VII. Assessment of the Nature of Water Relations Changes in Diseased Plants.- 7 Disease-Induced Alterations in Carbohydrate Metabolism.- I. Introduction.- II. Disease-Induced Alterations in Photosynthesis.- III. Mechanism of Disease-Induced Alterations in Photosynthesis.- IV. Disease-Induced Alterations in Transport of Photoassimilates.- V. Mechanism of Disease-Induced Alterations in Photoassimilate Transport.- VI. Disease-Induced Increases in Respiration.- VII. Mechanism of Disease-Induced Increases in Respiration.- VIII. Regulation of Carbohydrate Metabolism.- IX. Significance of Disease-Induced Alterations in Respiration.- X. Future Studies.- 8 Pathological Alterations in Transcription and Translation.- I. Introduction.- II. Disease-Induced Alterations in Transcription.- III. Disease-Induced Alterations in Translation.- IV. Conclusions.- 9 Alterations in Phenol Metabolism Caused by Disease.- I. Introduction.- II. Biosynthetic Pathways of Phenolic Compounds.- III. Disease-Induced Alterations in Phenol Metabolism.- 10 Growth Regulator Imbalance in Plant Disease.- I. Introduction.- II. Known Growth Regulators.- III. Pathogen-Produced Toxins with Growth Regulator Activity.- IV. Involvement of Growth Regulators in Certain Disease-Induced Abnormalities.- V. Mechanisms of Change in Indoleacetic Acid Content of Diseased Tissue.- VI. Influence of Growth Regulators on the Outcome of Disease Reaction.- VII. Limitations of Growth Regulator Studies.- VIII. Discussion.- 11 Crown Gall Tumor Formation.- I. Introduction.- II. The Bacterium.- III. Physiology of Crown Gall Tumor.- IV. Tumor Development.- V. Relations among Bacterial Plasmids.- VI. Mechanism of Transformation.- VII. Identification of Plasmid and Chromosomal Genes Involved in Oncogenicity.- VIII. Tumor Reversal.- 12 Mechanism of Disease Resistance.- I. Introduction.- II. Preformed Resistance Factors—Structural Features.- III. Preformed Resistance Factors—Preformed Antimicrobial Substances.- IV. Infection-Induced Resistance Factors—Physical Features.- V. Infection-Induced Resistance Factors—Chemical Features.- VI. The Dynamic and Coordinated Nature of Resistance.- 13 Induced Resistance.- I. Introduction.- II. Induced Resistance to Fungi.- III. Induced Resistance to Bacteria.- IV. Induced Resistance to Viruses.- V. Induced Resistance to Viroids.- VI. Some Features of Induced Resistance.- 14 Specificity in Plant-Pathogen Interactions.- I. Introduction.- II. Mechanisms o